Frederick Eberhardt

Patrick Burauel, Frederick Eberhardt, Michel Besserve

Unmeasured confounding is a major challenge for identifying causal relationships from non-experimental data. Here, we propose a method that can accommodate unmeasured discrete confounding. Extending recent identifiability results in deep latent variable models, we show theoretically that confounding can be detected and corrected under the assumption that the observed data is a piecewise affine transformation of a latent Gaussian mixture model and that the identity of the mixture components is confounded. We provide a flow-based algorithm to estimate this model and perform deconfounding. Experimental results on synthetic and real-world data provide support for the effectiveness of our approach.

Milan Mossé, Kara Schechtman, Frederick Eberhardt et al.

A person is directly racially discriminated against only if her race caused her worse treatment. This implies that race is an attribute sufficiently separable from other attributes to isolate its causal role. But race is embedded in a nexus of social factors that resist isolated treatment. If race is socially constructed, in what sense can it cause worse treatment? Some propose that the perception of race, rather than race itself, causes worse treatment. Others suggest that since causal models require \textit{modularity}, i.e. the ability to isolate causal effects, attempts to causally model discrimination are misguided. This paper addresses the problem differently. We introduce a framework for reasoning about discrimination, in which race is a high-level \textit{abstraction} of lower-level features. In this framework, race can be modeled as itself causing worse treatment. Modularity is ensured by allowing assumptions about social construction to be precisely and explicitly stated, via an alignment between race and its constituents. Such assumptions can then be subjected to normative and empirical challenges, which lead to different views of when discrimination occurs. By distinguishing constitutive and causal relations, the abstraction framework pinpoints disagreements in the current literature on modeling discrimination, while preserving a precise causal account of discrimination.

Erik Jahn, Frederick Eberhardt, Leonard J. Schulman

Causal discovery algorithms typically recover causal graphs only up to their Markov equivalence classes unless additional parametric assumptions are made. The sizes of these equivalence classes reflect the limits of what can be learned about the underlying causal graph from purely observational data. Under the assumptions of acyclicity, causal sufficiency, and a uniform model prior, Markov equivalence classes are known to be small on average. In this paper, we show that this is no longer the case when any of these assumptions is relaxed. Specifically, we prove exponentially large lower bounds for the expected size of Markov equivalence classes in three settings: sparse random directed acyclic graphs, uniformly random acyclic directed mixed graphs, and uniformly random directed cyclic graphs.

Sander Beckers, Frederick Eberhardt, Joseph Y. Halpern

Scientific models describe natural phenomena at different levels of abstraction. Abstract descriptions can provide the basis for interventions on the system and explanation of observed phenomena at a level of granularity that is coarser than the most fundamental account of the system. Beckers and Halpern (2019), building on work of Rubenstein et al. (2017), developed an account of abstraction for causal models that is exact. Here we extend this account to the more realistic case where an abstract causal model offers only an approximation of the underlying system. We show how the resulting account handles the discrepancy that can arise between low- and high-level causal models of the same system, and in the process provide an account of how one causal model approximates another, a topic of independent interest. Finally, we extend the account of approximate abstractions to probabilistic causal models, indicating how and where uncertainty can enter into an approximate abstraction.

Zhalama, Jiji Zhang, Frederick Eberhardt et al.

In recent years the possibility of relaxing the so-called Faithfulness assumption in automated causal discovery has been investigated. The investigation showed (1) that the Faithfulness assumption can be weakened in various ways that in an important sense preserve its power, and (2) that weakening of Faithfulness may help to speed up methods based on Answer Set Programming. However, this line of work has so far only considered the discovery of causal models without latent variables. In this paper, we study weakenings of Faithfulness for constraint-based discovery of semi-Markovian causal models, which accommodate the possibility of latent variables, and show that both (1) and (2) remain the case in this more realistic setting.

Krzysztof Chalupka, Pietro Perona, Frederick Eberhardt

We present and evaluate the Fast (conditional) Independence Test (FIT) -- a nonparametric conditional independence test. The test is based on the idea that when $P(X \mid Y, Z) = P(X \mid Y)$, $Z$ is not useful as a feature to predict $X$, as long as $Y$ is also a regressor. On the contrary, if $P(X \mid Y, Z) \neq P(X \mid Y)$, $Z$ might improve prediction results. FIT applies to thousand-dimensional random variables with a hundred thousand samples in a fraction of the time required by alternative methods. We provide an extensive evaluation that compares FIT to six extant nonparametric independence tests. The evaluation shows that FIT has low probability of making both Type I and Type II errors compared to other tests, especially as the number of available samples grows. Our implementation of FIT is publicly available.

Krzysztof Chalupka, Frederick Eberhardt, Pietro Perona

We propose a method to classify the causal relationship between two discrete variables given only the joint distribution of the variables, acknowledging that the method is subject to an inherent baseline error. We assume that the causal system is acyclicity, but we do allow for hidden common causes. Our algorithm presupposes that the probability distributions $P(C)$ of a cause $C$ is independent from the probability distribution $P(E\mid C)$ of the cause-effect mechanism. While our classifier is trained with a Bayesian assumption of flat hyperpriors, we do not make this assumption about our test data. This work connects to recent developments on the identifiability of causal models over continuous variables under the assumption of "independent mechanisms". Carefully-commented Python notebooks that reproduce all our experiments are available online at http://vision.caltech.edu/~kchalupk/code.html.

Krzysztof Chalupka, Tobias Bischoff, Pietro Perona et al.

We show that the climate phenomena of El Nino and La Nina arise naturally as states of macro-variables when our recent causal feature learning framework (Chalupka 2015, Chalupka 2016) is applied to micro-level measures of zonal wind (ZW) and sea surface temperatures (SST) taken over the equatorial band of the Pacific Ocean. The method identifies these unusual climate states on the basis of the relation between ZW and SST patterns without any input about past occurrences of El Nino or La Nina. The simpler alternatives of (i) clustering the SST fields while disregarding their relationship with ZW patterns, or (ii) clustering the joint ZW-SST patterns, do not discover El Nino. We discuss the degree to which our method supports a causal interpretation and use a low-dimensional toy example to explain its success over other clustering approaches. Finally, we propose a new robust and scalable alternative to our original algorithm (Chalupka 2016), which circumvents the need for high-dimensional density learning.

Antti Hyttinen, Sergey Plis, Matti Järvisalo et al.

This paper focuses on causal structure estimation from time series data in which measurements are obtained at a coarser timescale than the causal timescale of the underlying system. Previous work has shown that such subsampling can lead to significant errors about the system's causal structure if not properly taken into account. In this paper, we first consider the search for the system timescale causal structures that correspond to a given measurement timescale structure. We provide a constraint satisfaction procedure whose computational performance is several orders of magnitude better than previous approaches. We then consider finite-sample data as input, and propose the first constraint optimization approach for recovering the system timescale causal structure. This algorithm optimally recovers from possible conflicts due to statistical errors. More generally, these advances allow for a robust and non-parametric estimation of system timescale causal structures from subsampled time series data.

Krzysztof Chalupka, Pietro Perona, Frederick Eberhardt

We present a domain-general account of causation that applies to settings in which macro-level causal relations between two systems are of interest, but the relevant causal features are poorly understood and have to be aggregated from vast arrays of micro-measurements. Our approach generalizes that of Chalupka et al. (2015) to the setting in which the macro-level effect is not specified. We formalize the connection between micro- and macro-variables in such situations and provide a coherent framework describing causal relations at multiple levels of analysis. We present an algorithm that discovers macro-variable causes and effects from micro-level measurements obtained from an experiment. We further show how to design experiments to discover macro-variables from observational micro-variable data. Finally, we show that under specific conditions, one can identify multiple levels of causal structure. Throughout the article, we use a simulated neuroscience multi-unit recording experiment to illustrate the ideas and the algorithms.

Krzysztof Chalupka, Pietro Perona, Frederick Eberhardt

We provide a rigorous definition of the visual cause of a behavior that is broadly applicable to the visually driven behavior in humans, animals, neurons, robots and other perceiving systems. Our framework generalizes standard accounts of causal learning to settings in which the causal variables need to be constructed from micro-variables. We prove the Causal Coarsening Theorem, which allows us to gain causal knowledge from observational data with minimal experimental effort. The theorem provides a connection to standard inference techniques in machine learning that identify features of an image that correlate with, but may not cause, the target behavior. Finally, we propose an active learning scheme to learn a manipulator function that performs optimal manipulations on the image to automatically identify the visual cause of a target behavior. We illustrate our inference and learning algorithms in experiments based on both synthetic and real data.

Antti Hyttinen, Patrik O. Hoyer, Frederick Eberhardt et al.

We present a very general approach to learning the structure of causal models based on d-separation constraints, obtained from any given set of overlapping passive observational or experimental data sets. The procedure allows for both directed cycles (feedback loops) and the presence of latent variables. Our approach is based on a logical representation of causal pathways, which permits the integration of quite general background knowledge, and inference is performed using a Boolean satisfiability (SAT) solver. The procedure is complete in that it exhausts the available information on whether any given edge can be determined to be present or absent, and returns "unknown" otherwise. Many existing constraint-based causal discovery algorithms can be seen as special cases, tailored to circumstances in which one or more restricting assumptions apply. Simulations illustrate the effect of these assumptions on discovery and how the present algorithm scales.

Antti Hyttinen, Frederick Eberhardt, Patrik O. Hoyer

Much of scientific data is collected as randomized experiments intervening on some and observing other variables of interest. Quite often, a given phenomenon is investigated in several studies, and different sets of variables are involved in each study. In this article we consider the problem of integrating such knowledge, inferring as much as possible concerning the underlying causal structure with respect to the union of observed variables from such experimental or passive observational overlapping data sets. We do not assume acyclicity or joint causal sufficiency of the underlying data generating model, but we do restrict the causal relationships to be linear and use only second order statistics of the data. We derive conditions for full model identifiability in the most generic case, and provide novel techniques for incorporating an assumption of faithfulness to aid in inference. In each case we seek to establish what is and what is not determined by the data at hand.

Frederick Eberhardt, Clark Glymour, Richard Scheines

We show that if any number of variables are allowed to be simultaneously and independently randomized in any one experiment, log2(N) + 1 experiments are sufficient and in the worst case necessary to determine the causal relations among N >= 2 variables when no latent variables, no sample selection bias and no feedback cycles are present. For all K, 0 < K < 1/(2N) we provide an upper bound on the number experiments required to determine causal structure when each experiment simultaneously randomizes K variables. For large N, these bounds are significantly lower than the N - 1 bound required when each experiment randomizes at most one variable. For kmax < N/2, we show that (N/kmax-1)+N/(2kmax)log2(kmax) experiments aresufficient and in the worst case necessary. We over a conjecture as to the minimal number of experiments that are in the worst case sufficient to identify all causal relations among N observed variables that are a subset of the vertices of a DAG.

Frederick Eberhardt

We conjecture that the worst case number of experiments necessary and sufficient to discover a causal graph uniquely given its observational Markov equivalence class can be specified as a function of the largest clique in the Markov equivalence class. We provide an algorithm that computes intervention sets that we believe are optimal for the above task. The algorithm builds on insights gained from the worst case analysis in Eberhardt et al. (2005) for sequences of experiments when all possible directed acyclic graphs over N variables are considered. A simulation suggests that our conjecture is correct. We also show that a generalization of our conjecture to other classes of possible graph hypotheses cannot be given easily, and in what sense the algorithm is then no longer optimal.

Antti Hyttinen, Frederick Eberhardt, Patrik O. Hoyer

Given a set of experiments in which varying subsets of observed variables are subject to intervention, we consider the problem of identifiability of causal models exhibiting latent confounding. While identifiability is trivial when each experiment intervenes on a large number of variables, the situation is more complicated when only one or a few variables are subject to intervention per experiment. For linear causal models with latent variables Hyttinen et al. (2010) gave precise conditions for when such data are sufficient to identify the full model. While their result cannot be extended to discrete-valued variables with arbitrary cause-effect relationships, we show that a similar result can be obtained for the class of causal models whose conditional probability distributions are restricted to a `noisy-OR' parameterization. We further show that identification is preserved under an extension of the model that allows for negative influences, and present learning algorithms that we test for accuracy, scalability and robustness.