Ding Zhu

Ding Zhu, Vishnu Kabir Chhabra, Mohammad Mahdi Khalili

Cardiovascular disease is a major life-threatening condition that is commonly monitored using electrocardiogram (ECG) signals. However, these signals are often contaminated by various types of noise at different intensities, significantly interfering with downstream tasks. Therefore, denoising ECG signals and increasing the signal-to-noise ratio is crucial for cardiovascular monitoring. In this paper, we propose a deep learning method that combines a one-dimensional convolutional layer with transformer architecture for denoising ECG signals. The convolutional layer processes the ECG signal by various kernel/patch sizes and generates an embedding called multi-scale patch embedding. The embedding then is used as the input of a transformer network and enhances the capability of the transformer for denoising the ECG signal.

Vishnu Kabir Chhabra, Ding Zhu, Mohammad Mahdi Khalili

Previous research has shown that fine-tuning language models on general tasks enhance their underlying mechanisms. However, the impact of fine-tuning on poisoned data and the resulting changes in these mechanisms are poorly understood. This study investigates the changes in a model's mechanisms during toxic fine-tuning and identifies the primary corruption mechanisms. We also analyze the changes after retraining a corrupted model on the original dataset and observe neuroplasticity behaviors, where the model relearns original mechanisms after fine-tuning the corrupted model. Our findings indicate that: (i) Underlying mechanisms are amplified across task-specific fine-tuning which can be generalized to longer epochs, (ii) Model corruption via toxic fine-tuning is localized to specific circuit components, (iii) Models exhibit neuroplasticity when retraining corrupted models on clean dataset, reforming the original model mechanisms.