Andrew Herren

2papers

2 Papers

MLMay 6, 2024
Deep Learning for Causal Inference: A Comparison of Architectures for Heterogeneous Treatment Effect Estimation

Demetrios Papakostas, Andrew Herren, P. Richard Hahn et al.

Causal inference has gained much popularity in recent years, with interests ranging from academic, to industrial, to educational, and all in between. Concurrently, the study and usage of neural networks has also grown profoundly (albeit at a far faster rate). What we aim to do in this blog write-up is demonstrate a Neural Network causal inference architecture. We develop a fully connected neural network implementation of the popular Bayesian Causal Forest algorithm, a state of the art tree based method for estimating heterogeneous treatment effects. We compare our implementation to existing neural network causal inference methodologies, showing improvements in performance in simulation settings. We apply our method to a dataset examining the effect of stress on sleep.

MESep 14, 2020
Semi-supervised learning and the question of true versus estimated propensity scores

Andrew Herren, P. Richard Hahn

A straightforward application of semi-supervised machine learning to the problem of treatment effect estimation would be to consider data as "unlabeled" if treatment assignment and covariates are observed but outcomes are unobserved. According to this formulation, large unlabeled data sets could be used to estimate a high dimensional propensity function and causal inference using a much smaller labeled data set could proceed via weighted estimators using the learned propensity scores. In the limiting case of infinite unlabeled data, one may estimate the high dimensional propensity function exactly. However, longstanding advice in the causal inference community suggests that estimated propensity scores (from labeled data alone) are actually preferable to true propensity scores, implying that the unlabeled data is actually useless in this context. In this paper we examine this paradox and propose a simple procedure that reconciles the strong intuition that a known propensity functions should be useful for estimating treatment effects with the previous literature suggesting otherwise. Further, simulation studies suggest that direct regression may be preferable to inverse-propensity weight estimators in many circumstances.